In the production of gold and silver cyanide process, when escaping from the inflatable primary toxic cyanide cyanide solution, heating and acidizing HCN, and the solid dust solution containing cyanide and cyanogen. In industrial production, cyanide enters the body mainly through the respiratory tract and skin, and cyanide dust can also be inhaled through the digestive tract. HCN breaks down cyanide in the air or in the human body. Cyanide is a highly toxic substance. Taking sodium cyanide as an example, oral inhalation of 50 to 60 mg (calculated as cyanogen) can cause death in an instant. Fish can be killed by taking 0.04~0.1mg∕kg body weight. The toxic effect of cyanide is that after cyanide ions enter the human body, the water in the body is rapidly decomposed and combined to form HCN. In particular, cyanide ions can rapidly bind to the trivalent iron of oxidized cytochrome oxidase, and inhibit the reduction of cytochrome to produce reduced cytochrome oxidase with ferrous iron, thereby inhibiting the oxidation of cytochrome and preventing tissue cells from being timely. Sufficient oxygen is obtained, causing the biooxidation to not proceed normally, and intracellular compartments occur. Because the central nervous system is most sensitive to hypoxia, the brain is firstly damaged. Respiratory central paralysis is often the cause of death from cyanide poisoning. Therefore, after inhaling a high concentration of HCN, it can cause respiratory arrest and death within 2 to 3 minutes. The cyanide-containing wastewater discharged from the factory will pollute the rivers and lakes in a large area, and the consequences are extremely serious. China's "Integrated Wastewater Discharge Standard (GB8978-88)" stipulates that: the discharge into the urban centralized drinking water area, economic fishery waters, scenic tourist areas, industrial and agricultural water areas, ports, etc., the maximum allowable discharge concentration of cyanide 0.5mg ∕L The maximum allowable discharge concentration is 1.0mg∕L when discharged into the urban sewer and entering the secondary sewage treatment plant. Cyanide poisoning is mainly acute poisoning, and clinical symptoms can be divided into four phases: First, the precursor period. Irritations, burning, numbness, vomiting in the upper respiratory tract such as eyes and throat, accompanied by dizziness, headache, tinnitus, fatigue, and tight stools. Second, the period of difficulty breathing. Chest tightness, palpitations, shortness of breath, elevated blood pressure, rapid pulse, and irregular heart rhythm, gradually became unconscious and entered a coma. Third, the flood season. Hemorrhoids, convulsions, incontinence, sweating and decreased body temperature. Fourth, the paralysis period. Feelings and reflections disappear, breathing slows and stops. In severe cases of acute cyanide poisoning, if it can be rescued promptly and properly, it may stop the development of the disease and make the poisoned person hope to be rescued. After the mild poisoning of acute poisoning, it can gradually improve and recover after 2 to 3 days. Chronic poisoning caused by long-term inhalation can cause neurasthenia, general malaise, neuromuscular pain and gastrointestinal symptoms. Poisoning from skin contact can cause macules, papules and herpes. Potassium cyanide can cause small blemishes and blisters in contact with the skin. Since the detoxification of cyanide into the human body is mainly exhaled by the lungs in the form of HCN, HCN has a almond flavor. Therefore, in addition to the history of exposure to cyanide, the diagnosis of cyanide poisoning has a certain reference value in the exhalation with almond flavor. In view of the acute toxicity of cyanide, production workshops should be based on prevention. The main measures to prevent cyanosis include: 1. Prevent solid cyanide dust from contaminating hands, face, clothes, tables, chairs, and floors. Bathe and change clothes after work. In particular, dust is prevented from being inhaled by the mouth. Second, the equipment or workshop that produces HCN gas is best sealed, and the air containing HCN is extracted by the exhaust fan and washed with dilute alkali solution and then emptied. 3. Cyanide-containing wastewater and washing water can be discharged after being treated to the standard. Fourth, reform the production process, try to use mechanized or automated feeding to reduce direct contact with people. 5. The production workshop should be equipped with emergency medicine and equipment. The operator should not only know how to prevent poisoning, but also should be familiar with the rescue method of poisoning, so as to timely rescue on the spot. Currently, the combination therapy of nitrite and thiosulfate is mainly used for the treatment of acute cyanide poisoning. It includes the following rescue process: 1. Dissolve isoamyl nitrite (an ampoule) in a handkerchief and repeat the inhalation of the poisoned person for 1 to 2 times in 15 minutes, each time for 15 to 30 seconds. Second, at a rate of 2 to 3 mL per minute, slowly inject 3% sodium nitrite 10-20 mL. A too fast injection rate will cause a sudden drop in blood pressure. Third, intravenous injection of 25% to 50% sodium thiosulfate 25 ~ 50mL. If necessary, re-inject half or full amount within 1 hour. Since glucose can bind to HCN to form a non-toxic nitrile, it can be used simultaneously when injecting the above drugs. Fourth, according to animal experiments show that, depending on the cobalt acid effective in treating cyanide poisoning. When the condition is critical, an intravenous drip can be added to the glucose solution with a dose of 600 mg. When the skin is hydrogen cyanide burns, rinse with micros potassium permanganate solution, washed with ammonium sulfide solution. Iron Box,Iron Boxes,Iron Box Packaging,Food Packaging Dongguan Tielangtou Hardware Products Co., Ltd , https://www.tinboxtlt.com